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Rowena N. Schwartz, Pharm.D., BCOP, Program Moderator Associate Professor, Pharmacy and Therapeutics School of Pharmacy University of Pittsburgh Pittsburgh, Pennsylvania Val R. Adams, Pharm.D., FCCP, BCOP Associate Professor, Pharmacy Practice and Science College of Pharmacy University of Kentucky Lucille P. Markey Cancer Center Lexington, Kentucky. The usual therapeutic range for monotherapy is 10 to mg bromocriptine a day. 1. Kanter SJ, Factora RM, Suh TT. Does this patient have primary progressive aphasia? Cleve Clin J Med 2006; 73: 10251027. Reed DA, Johnson NA, Thompson C, Weintraub S, Mesulam MM. A clinical trial of bromocriptine for treatment of primary progressive aphasia Letter ; . Ann Neurol 2004; 56: 750.

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Rate, and body position. Patients found to have OSA then underwent an unattended CPAP titration using an auto-titrating CPAP device. After 30 days of treatment, the questionnaires and PVT were repeated. Statistical analyses were performed and statistical significance was defined as 2-tail alpha value of 0.05. Primary outcomes measured were CPAP use and PVT scores. Subjective measures included perception of treatment efficacy 6-point Likert scale ; , sleepiness by ESS scores, and QOL by SF-36. Results : Fourteen male subjects, age 37-75 years mean 48.57, S.D. 9.83 ; completed the study. Apnea Hypopnea Index before treatment was 44.62 events per hour S.D. 24.13 ; . Quality of life as measured by SF-36 improved significantly for physical health, general health, vitality, social functioning, emotional and mental health subscales. Physical function trended towards improvement. Epworth scores improved from 13.21 S.D.4.08 ; to 5.57 S.D. 3.78 ; after treatment. Compliance was available for 8 subjects with mean use of 4.04 hours nightly range 0 - 7.45 hrs ; . PVT pretreatment mean 351.05, S.D. 175.94; posttreatment mean 304.58, S.D. 102.53 ; showed a trend towards improvement Conclusion : In selected patients, ambulatory management of obstructive sleep apnea improved the patient's QOL. These improvements are comparable to previously published results using traditional in-laboratory management. Support optional ; : This study received support by the ResMed Research Foundation, for example, side effects of bromocriptine.
Yuen BH, Cannon W, Sy L, Booth J, et al. Regression of pituary microadenoma during and following bromocriptine therapy: persistent defect in prolactin regulation before and throughout pregnancy. J Obstet Gynecol 1982; 142: 634-639. Yukiyama S, Shinomiya M, Ikebuchi K, Sato T. Reproductive and developmental toxicity study of a new antineoplastic agent, S-1 II ; - Teratological study in rats by oral administration. J Toxicol Sci 1996; 21 S3 ; : 603-618. Yunis KA, Bitar FF, Hayek P, et al. Trancient hypertonic cardiomyopathy in the newborn following multiple doses. Current dopamine agonists include bromocriptine parlodel ; , pergolide permax ; , cabergoline cabaser ; , lisuride dopergin revanil ; , apomorphine apokyn ; , pramipexole mirapex ; , ropinirole requip ; and rotigotine neupro and cabergoline. The half-life of parent drug is fourfold higher in patients with renal failure table 1 ; 23.
Complications and Fluctuations of Long-term Treatment Unfortunately drug side effects from long term use of medications may become worse than the symptoms they're meant to suppress: a ; Dyskinesia This means bizarre, involuntary, jerky movements of the head, tongue, and extremities, and is a particularly troublesome side-effect of L-dopa. The abnormal movements can gradually become incapacitating unless the L-dopa dosage is reduced. Small doses of L-dopa, stopping anticholinergics, using bromocriptine with L-dopa, and Clozapine may help to control this. b ; "Frozen State" A wearing off response to medication, with a return of symptoms can produce an awkward, "frozen" state that may persist until more medication is absorbed, although people sometimes spontaneously "unfreeze" without more medication. Freezing episodes, especially when starting to walk, run, or change direction, may become frequent as the effect of the drugs wear off. For some people taking more medication can bring on bad side effects, but not taking it may mean a worsening of the Parkinsonian symptoms. c ; End of dose deterioration As each dose of medication wears off, symptoms may return with varying "good" and "bad" times through the day. More frequent L-dopa doses, sometimes with bromocriptine may help, and Sinemet CR may help. d ; Addiction to L-dopa People who have had a problem with drug addiction alcohol, street drugs ; may become addicted to L-dopa, and experience withdrawal effects when it has to be stopped. e ; The "On-Off" Phenomenon Sudden spells of immobility, apparently unrelated to drug doses, may occur several times a day and last from minutes to hours. Drugs do not help this type of parkinsonian immobility, but lowering the L-dopa dosage and sometimes adding bromocriptine ; may help these. Conditions Influencing Treatment People with PS often have other medical conditions. Sometimes these conditions, or the drugs that are prescribed for them, influence the treatment of PS. Some of these conditions are and cafergot. Ovulation and pregnancy rate Normalisation of prolactin: Cabergoline: 56 60 93.3% ; Adverse effect Bromocriptine: 27 58 52% ; RR 2.0 CI 1.51 to 2.66 ; Gastrointestinal symptoms Ovulation and pregnancy rate; Cabergoline: 43 60 71.6% ; Bromocriptine: 28 58 48.2% ; RR 1.48 CI 1.09 to 2.02 ; Adverse effects: Cabergoline: 32 60 53.3% ; Bromocriptine: 38 58 65.5% ; RR 0.81 CI 0.60 to 1.10 ; Gastrointestinal symptoms: Cabergoline: 22 60 36.6% ; Bromocriptine: 49 58 84.5% ; RR 0.43 CI 0.31 to 0.62.

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NDC 00781220105 00781220201 00781220205 Label Name TEMAZEPAM 15MG CAPSULE TEMAZEPAM 30MG CAPSULE TEMAZEPAM 30MG CAPSULE TEMAZEPAM 7.5MG CAPSULE FIORTAL CODEINE #3 CAPSULE THIOTHIXENE 1MG CAPSULE THIOTHIXENE 2MG CAPSULE THIOTHIXENE 2MG CAPSULE THIOTHIXENE 5MG CAPSULE THIOTHIXENE 5MG CAPSULE THIOTHIXENE 10MG CAPSULE THIOTHIXENE 10MG CAPSULE HYDROXYZINE PAM 25MG CAP HYDROXYZINE PAM 25MG CAP HYDROXYZINE PAM 50MG CAP DOCUSATE SODIUM 100MG CAP DOCUSATE SODIUM 100MG CAP NITROFURANTOIN MCR 50MG CAP NITROFURANTOIN MCR 50MG CAP NITROFURANTOIN MCR 50MG CAP NITROFURANTOIN MCR 100MG CP TRIAMTERENE HCTZ 50 25 CAP TRIAMTERENE HCTZ 50 25 CAP DOCUSATE CALCIUM 240MG CAP AMOXICILLIN 500MG CAPSULE NORTRIPTYLINE HCL 10MG CAP NORTRIPTYLINE HCL 25MG CAP NORTRIPTYLINE HCL 25MG CAP NORTRIPTYLINE HCL 50MG CAP NORTRIPTYLINE HCL 75MG CAP DSS CASANTHRANOL 100 30 CAP DSS CASANTHRANOL 100 30 CAP TRIAMTERENE HCTZ 50 25 CAP TRIAMTERENE HCTZ 50 25 CAP DOCUSATE SODIUM 250MG CAP OXAZEPAM 10MG CAPSULE OXAZEPAM 15MG CAPSULE OXAZEPAM 15MG CAPSULE OXAZEPAM 30MG CAPSULE BROMOCRIPTINE 5MG CAPSULE BROMOCRIPTINE 5MG CAPSULE FLUOXETINE 20MG CAPSULE FLUOXETINE 10MG CAPSULE FLUOXETINE 40MG CAPSULE URSODIOL 300MG CAPSULE RANITIDINE 150MG CAPSULE RANITIDINE 150MG CAPSULE RANITIDINE 300MG CAPSULE RANITIDINE 300MG CAPSULE PSEUDO-CHLOR CAPSULE SA DOXAZOSIN MESYLATE 1MG TAB DOXAZOSIN MESYLATE 2MG TAB DOXAZOSIN MESYLATE 2MG TAB No. Claims 1, 558 1, Amount Paid $14, 257.16 $10, 924.67 $7, 681.65 $46, 013.94 $7, 402.30 $567.75 $2, 723.30 $616.14 $7, 092.73 $353.02 $8, 246.41 $1, 016.35 $529.60 $29.08 $537.15 $7, 592.08 $1, 275.29 $23, 881.50 $4, 426.54 $1, 058.73 $20, 760.11 $1, 867.82 $2, 250.96 $14, 348.13 $17, 001.26 $1, 408.87 $3, 452.40 $568.49 $3, 300.26 $1, 271.10 $1, 711.67 $6.01 $9, 665.54 $6, 814.77 $168.69 $1, 552.06 $4, 074.85 $319.56 $5, 740.14 $1, 619.77 $297.95 $25, 517.68 $890, 136.43 $14, 703.15 $7, 722.79 $418, 126.48 $75, 312.27 $7, 403.89 $8, 924.91 $666.54 $5, 819.94 $12, 477.99 $28.98 and calan.

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Table 2.1: The properties of the ideal alpha helix. Phi, psi, and omega are the torsion angles, refer back to Figure 2.3 on page 11 for reference if necessary. The translation is the distance in ngstrms along the helical axis moved per A o residue [PR04]. Alpha helices are easily distorted, a fact that makes their study non-trivial. The axis of a helix is never a straight line, as helices are always bent to some degree. This is often attributable to steric interactions with other regions of the protein or. Other potential targets for antidepressants are cytokines, neurokinins and N-methyl-D-aspartate NMDA ; receptors. A number of alterations of the immune system are observed in major depression. Particularly increased levels of proinflammatory cytokines can be detected Maes et al., 1997 ; . It is unclear, however, whether this is a consequence of depression or a causal effect. In favour of a causal relationship, treatments with interleukin IL ; -2 and interferon INF ; - used in patients with hepatitis C and some cancer forms frequently produce depressive symptoms Schaefer et al., 2002 ; . After chronic treatment with antidepressant drugs, serum IL-1 and serum IL-6 are reduced to normal levels in depressed patients Sluzewska et al., 1996 ; . In one study, an increased production of IL-1-receptor antagonist mRNA in specific regions of the rat cortex could be detected Suzuki et al., 1996 ; . This raises the possibility that drugs normalising the levels of cytokines IL-1, IL-6 ; and IL-1-receptor antagonists could be a novel therapeutic strategy for the treatment of depression, but possibly the side-effects may outweigh the antidepressant effect. Clinical trials demonstrated that neurokinin-1 NK-1 ; receptor antagonists were surprisingly effective in treating depression Stout et al., 2001 ; and a selective NK-2-receptor antagonist was shown to display antidepressant-like properties in and capoten.

Aggregate Family Deductible A deductible which is met when two or more family members' charges are added together to meet a family deductible. Allergy Treatment The treatment of the allergic patient may include identifying the offending agent by means of various testing methods. Once the agent is identified, treatment is provided by avoidance, medication, or immunotherapy. Allowable Charge The maximum fee that a health plan will reimburse a provider for a given service. Allowance The amount an individual provider member is entitled to receive for a certain service. Allowed Amount The maximum reimbursement the member's health policy allows for a specific service in or out of network. This amount may be: -a fee negotiated with participating providers -a customary charge based on the amount -charged by most providers in the member's area -an allowance established by law -an amount set on a Fee Schedule of Allowance Ambulatory Care All types of health services that are provided on an outpatient basis. Ambulatory Care Facility A medical care center that provides a wide range of healthcare services, including preventive care, acute care, surgery and outpatient care in a centralized facility. Ambulatory Surgery Surgical procedures performed that do not require an overnight hospital stay. Procedures can be performed in a hospital or a licensed surgical center. Also called Outpatient Surgery. Ancillary Services Auxiliary or supplemental services i.e. diagnostic services, physical therapy, medications ; used to support diagnosis and treatment of a patient's condition. Annual out-of-pocket Coinsurance Maximum The most you will have to pay in out-of-pocket costs for coinsurance on covered services during a calendar year. Charges in excess of the allowed amount see definition ; are not applied toward this maximum. Appeal s ; A process used by a provider or member to request the health plan reconsider a previous authorization or claim decision. Assignment An agreement in which a patient assigns to another party, usually a physician or hospital, the right to receive payment from a public or private insurance program for the service the patient has received. Attending Physician Physician primarily responsible for the care of a patient during hospitalization. Authorization See 'Pre-Authorization' Authorized Services See 'Precertified Services'. Environment The risk that KOH poses for the environment is essentially restricted to a pH increase of the aquatic compartment, which is dependent on the hardness of the waters. This effect is well known, as are the ways to control it. Therefore, no further testing is required. Human health The risk that KOH poses for human health originates from its corrosive properties essentially. These are sufficiently documented in order to take the indicated precautions of use. Therefore no further testing is required and carbidopa. W ithin 90 days after a scientific advisory panel makes recommendations on any matter under its review, the Food and Drug Adm inistration official responsible for the matter shall review the conclusions and recommend ations of the panel, and notify the affected persons of the final decision on the matter, or of the reasons that no such decision has been reached. Each such final decision shall be documented including the rationale for the decision, because bromocriptine parkinsons.

Onist flumazenil Romazicon ; may improve mental status transiently, whereas br0mocriptine Parlodel ; may improve extrapyramidal symptoms.13 No formal recommendation for the routine use of any of these agents has been suggested. Regardless of the etiology of cirrhosis, the development of portal hypertension is nearly universal and results from an increased resistance to portal flow secondary to scarring, narrowing, and compression of the hepatic sinusoids. When the portal pressure exceeds a certain threshold, it results in the development of varices. Approximately 50 percent of patients with cirrhosis develop varices, most commonly in the distal 2 to 5 the esophagus.14 Variceal hemorrhage is defined as bleeding from an esophageal or gastric varix at the time of endoscopy, or the presence of large esophageal varices with blood in the stomach and no other recognizable source of bleeding.9 The rate of variceal bleeding is approximately 10 to 30 percent per year.14 The British Society of Gastroenterology guidelines for the management of variceal hemorrhage recommend and levodopa.
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With MRI findings of Brain suggestive of deep cerebral veins thrombosis causing venous infarct in bilateral thalami, left caudate and left lentiform nuclei.3 Patient was started on bromocriptine. Within a period of ten days, patient improved. She was able to imitate some speech limited to her needs, she started responding to others in complete sentences, eating a full diet orally, participating in conversations and jokes and was discharged from the hospital. At two months post stroke, patient had recovered well. However she could not resume complete household responsibilities. She showed mild memory deficits and goal directed behavior was limited to smaller tasks. Larger tasks involving several steps were daunting e.g. cooking an elaborate meal required several external prompts. This memory disturbance can be attributed to thalamic involvement.4.

Although for the most part bromocrlptine mesylate will not be used or be beneficial for women in a bodybuilding strength athletics sense, the drug itself has been found not to be harmful to women and carvedilol. She prescribed the one drug that is safe.

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Musculoskeletal: Myopathy with elevated CPK plus muscle pain, weakness or tenderness fever and malaise. Rhabdomyolysis with renal failure reported. Hepatic: Elevated transaminases in 1% to 2%; discontinue if ALT and or AST shows unexplained increase 3x upper limit of normal ULN ; x 2. 6 Drugs: Atorvostatin Miscellaneous: Diarrhea, constipation, nausea, heartburn, stomach pain, dizziness, headache, skin rash, impotence rare ; , insomnia and cilostazol. State and School Employees' Life and Health Insurance Plan State and School Employees Health Insurance Management Board c o Department of Finance and Administration P.O. Box 24208 Jackson, MS 39225-4208 January 1, 2004 through December 31, 2004.

Or lymphocytic alveolitis has been observed [9, 12], and recently two definite cases of interstitial pneumonitis due to dihydroergocristine have been described [13]. Apart from pleural calcifications, which are lacking in ergot-induced pleurisy, ergot-induced pleural lesions are indistinguishable from those induced by asbestos [13]. Treatment consists of withdrawal of the offending drug, and improvement of systemic features thereafter is the best diagnostic test. The outcome is most often favourable, but complete recovery seems to be rare [7, 10, 13]. It has to be stressed that pleural disease may progress and cause serious illness if the condition remains unrecognized [10]. One of the questions that these case reports raise is: do pre-existing asbestos-induced pleural lesions increase the risk of developing drug-induced pleural disease? Bromocriptine-induced pleural effusion has previously been seen in an asbestos-exposed machine operator [9] as well as in an asbestos-exposed ironer [13]. Moreover, an increased risk of ergot derivative-induced pleural disease in patients with asbestos-related disease has recently been reported by HILLERDAL et al. [14]. This suggests that patients with asbestos pleural plaques, or even asbestos exposure, but without radiologically detectable lesions, may have an increased risk of developing druginduced pleural effusions. Mechanisms of interaction between asbestos fibres and ergoline drugs are unknown, but the two agents may act in concert to elicit pleural inflammation. It is also conceivable that the presence of pleural plaques and or asbestos fibres in the parietal pleura may block lymphatic structures involved in the drainage of pleural fluid [15]. In conclusion, although benign asbestos pleural effusion is a well-accepted clinical entity in asbestos-exposed workers, drug-induced effusion should be considered as well. A thorough work-up to determine the cause of pleural effusion in those taking ergolines is not only of academic or medicolegal interest, but also of practical therapeutic importance. Finally, although we cannot prove it, we feel that the association of asbestos- and bromocriptine-induced pleural disease in these patients was not fortuitous, but rather that patients exposed to asbestos may be at an increased risk of pleural effusions of any kind, which may relate to impaired fluid clearance from the pleural cavities and ciprofloxacin and bromocriptine.

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Bromocriptine is beneficial for various conditions such as cocaine addiction and mastalgia, although these are not fda-approved indications for the drug. The frequency of M tended P .06 ; to be lower in treated rams than in control rams during the 1st wk of treatment Figure 3B, wk 2 ; . During wk 3 and 4, M frequency was lower in bromocriptine-treated rams than in control rams P .05 ; . The tendency for lower M frequency persisted in treated rams after the withdrawal of bromocriptine Figure 3B, wk 6, P .2 ; . Frequency of ejaculation decreased in all rams throughout the experimental period in the spring. However, in wk 2 and 4, E frequency tended to be lower in bromocriptine-treated rams than in control rams P .09 ; Figure 3C ; . When the frequency of MA and M were summed, which eliminates confounding the sexual activity of rams with the variable receptivity of females MA and clarinex.

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If the request for prior authorization is approved, Managed Drug Limitation parameters will apply. * If the request for prior authorization is approved, drug is to be obtained through the Specialty Rx program. G. Injectable Medication through Specialty Rx Members on specialty injectable drugs must purchase them through AAHPS's specialty pharmaceutical vendor. Your prescribing physician will complete a referral form for these drugs and submit it to the specialty pharmacy vendor for processing and delivery to the member's home, the physician's office or to the location of their choice. For more information on this program and a list of medication available, please contact the Member Services Department at 706 ; 549-0549. H. Exclusions In addition to all of the Exclusions and Limitations contained in the Evidence of Coverage the following Exclusions apply to this rider: a. Experimental drugs or investigational drugs. b. Coverage of medications outside the HPS approved policy guidelines for coverage of the medication. c. Drugs or medicines purchased and received prior to the member's effective date or following the member's termination of coverage. AAHPS RxRider Rev 10 06 ; 6.
1. 2. 3. Lang AE, Lozano AM. Medical progress: Parkinson's Disease first of two parts ; . New England Journal of Medicine 1998; 339 15 ; : 1044-53. Lang AE, Lozano AM. Medical progress: Parkinson's disease second of two parts ; . New England Journal of Medicine 1998; 339 16 ; : 1130-43. Schapira AHV. Science, medicine, and the future: Parkinson's disease. British Medical Journal 1999; 318: 311-4. Gottwald MD, et al. New pharmacotherapy for Parkinson's disease. [Review] [59 refs]. Annals of Pharmacotherapy 1997; 31 10 ; : 1205-17. Lange KW. Clinical pharmacology of dopamine agonists in Parkinson's disease. [Review] [83 refs]. Drugs & Aging 1998; 13 5 ; : 381-9. Mirapexin, Summary of Product Characteristics. 1999; Pharmacia & Upjohn. Guttman M. Double-blind comparison of pramipexole and bromocriptine treatment with placebo in advanced Parkinson's disease. International Pramipexole-Bromocriptine Study Group. Neurology 1997; 49 4 ; : 1060-5. Lieberman A, et al. Clinical evaluation of pramipexole in advanced Parkinson's disease: results of a double-blind, placebo-controlled, parallel-group study. Neurology 1997; 49 1 ; : 162-8. Pogarell O, et al. A non-ergot dopamine agonist, pramipexole in the therapy of advanced Parkinson's disease; Improvement of Parkinsonian symptoms and treatment-associated complications. A review of three studies. Clinical Neuropharmacology 1997; 20 supplement 1 ; : S28-S35 Wermuth L, The Danish Pramipexole Study Group. A double-blind, placebo-controlled, randomized, multi-center study of pramipexole in advanced Parkinson's disease. European Journal of Neurology 1998; 5: 235-42. Pinter MM, et al. Efficacy, safety, and tolerance of the non-ergoline dopamine agonist pramipexole in the treatment of advanced Parkinson's disease: a double blind, placebo controlled, randomised, multicentre study. Journal of Neurology, Neurosurgery & Psychiatry 1999; 66 4 ; : 436-41. Shannon KM, et al. Efficacy of pramipexole, a novel dopamine agonist, as monotherapy in mild to moderate Parkinson's disease. The Pramipexole Study Group [published erratum appears in Neurology 1998 Mar; 50 3 ; : 838]. Neurology 1997; 49 3 ; : 724-8. Anonymous. Safety and efficacy of pramipexole in early Parkinson disease. A randomized dose-ranging study. Parkinson Study Group. JAMA 1997; 278 2 ; : 125-30. Hubble JP, et al. Pramipexole in patients with early Parkinson's disease. Clinical Neuropharmacology 1995; 18 4 ; : 338-47. Anonymous. Studies support dopamine agonist monotherapy. Scrip 1999; 2459 ; : 23 Personal communication, Pramipexole Mirapexin ; . Information, Pharmacia and Upjohn Limited. 1999. Medical.
Table 1. STDs among San Francisco residents, July, 2006. 2005 month YTD month Gonorrhea Male rectal gonorrhea Chlamydia Male rectal chlamydia LGV Syphilis adult total ; Primary & secondary Early latent Unknown latent Late latent Neurosyphilis Congenital syphilis PID Adult male shigellosis 244 64 341 0 42 0 177 0 39. Eli Lilly Lilly Pharma Fertigung Eli Lilly UCB s.a., Secteur Pharma UCB s.a., Secteur Pharma UCB s.a., Secteur Pharma Manufacture Assembly primary site ; QC testing Assembly alternative site ; Final Release Manufacture Assembly primary site ; QC testing Assembly alternative site ; Final Release Manufacture Assembly primary site ; QC testing Assembly alternative site ; Final Release Manufacture Assembly primary site ; QC testing Assembly alternative site ; Final Release, because bromocriptine mesylate. You should always use drugs under a physician's supervision and cabergoline. The first step in analyzing the respiration channels was to weigh and add the raw signals of the two bands to give an estimation of changes in instantaneous lung volume. Weighting or band stretch-to-volume ; coefficients were established by calibration procedures conducted before and after the flight. During this calibration subjects were instructed to first breathe primarily by moving their rib cage for 810 cycles and then breathe primarily by moving their abdomen Chadha et al., 1982 ; . The Respitrace signals were monitored to make sure that subjects could follow this instruction. If subjects had difficulty in separating the two kinds of breathing, they were briefly trained how to do so. A least-squares fit multiple regression computer program developed in our laboratory established band stretch-to-volume coefficients to best predict spirometer volume from output from the two bands. These coefficients were the mean of two calibrations, one at the beginning and one at the end of the experiment about 5 h apart ; . The two values showed high retest reliability: in this sample R 2 for the thoracic band was 0.92 and for the abdominal band, 0.83. Relative deviation of thoracic coefficients of one calibration from the mean of two calibrations was 4.1% S.D. 5.3%, range 0.0 24.7% ; . For abdominal coefficients these values were 8.9.
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